Anosmia, the loss of the sense of smell, is one of the main neurological manifestations of
COVID-19. Although the SARS-CoV-2 virus targets the nasal olfactory epithelium, current …

Numerous pathogens can infect the olfactory tract, yet the pandemic caused by SARS-CoV-
2 has strongly emphasized the importance of the olfactory mucosa as an immune barrier …

Background As a debilitating condition that can impact a whole spectrum of people and
involve multi-organ systems, long COVID has aroused the most attention than ever …

Background We have recently demonstrated a causal link between loss of gonadotropin-
releasing hormone (GnRH), the master molecule regulating reproduction, and cognitive …

With the decline in the number of new Severe Acute Respiratory Syndrome Coronavirus 2
(SARS‐CoV‐2) infections, the World Health Organization announced the end of the SARS …

Background As a consequence of SARS-CoV-2 infection various neurocognitive and
neuropsychiatric symptoms can appear, which may persist for several months post infection …

Neuroinvasion of SARS-CoV-2 is a matter of debate [5, 8, 10]. While some animal models (in
particular genetically modified ones) revealed a consistent and fatal neuroinvasion of SARS …

Infection, autoimmunity, and cancer are principal human health challenges of the 21 st
century. Often regarded as distinct ends of the immunological spectrum, recent studies hint …

The olfactory mucosa is a component of the nasal airway that mediates the sense of smell.
Recent studies point to an important role for the olfactory mucosa as a barrier to both …

Background SARS-CoV-2 is a single-stranded positive-sense RNA virus. Several negative-
sense SARS-CoV-2 RNA species, both full-length genomic and subgenomic, are produced …