Tumor necrosis factor receptor‐associated factor 6 (TRAF 6) regulation of development, function, and homeostasis of the immune system
MC Walsh, JE Lee, Y Choi - Immunological reviews, 2015 - Wiley Online Library
Tumor necrosis factor receptor (TNFR)‐associated factor 6 (TRAF 6) is an adapter protein
that mediates a wide array of protein–protein interactions via its TRAF domain and a RING …
that mediates a wide array of protein–protein interactions via its TRAF domain and a RING …
[HTML][HTML] TRAF molecules in cell signaling and in human diseases
P Xie - Journal of molecular signaling, 2013 - Springer
The tumor necrosis factor receptor (TNF-R)-associated factor (TRAF) family of intracellular
proteins were originally identified as signaling adaptors that bind directly to the cytoplasmic …
proteins were originally identified as signaling adaptors that bind directly to the cytoplasmic …
Genetic alterations of TRAF proteins in human cancers
S Zhu, J Jin, S Gokhale, AM Lu, H Shan… - Frontiers in …, 2018 - frontiersin.org
The tumor necrosis factor receptor (TNF-R)-associated factor (TRAF) family of cytoplasmic
adaptor proteins regulate the signal transduction pathways of a variety of receptors …
adaptor proteins regulate the signal transduction pathways of a variety of receptors …
The PERK arm of the unfolded protein response regulates satellite cell-mediated skeletal muscle regeneration
Regeneration of skeletal muscle in adults is mediated by satellite stem cells. Accumulation
of misfolded proteins triggers endoplasmic reticulum stress that leads to unfolded protein …
of misfolded proteins triggers endoplasmic reticulum stress that leads to unfolded protein …
TRAF6 regulates satellite stem cell self-renewal and function during regenerative myogenesis
Satellite cells are a stem cell population within adult muscle and are responsible for
myofiber regeneration upon injury. Satellite cell dysfunction has been shown to underlie the …
myofiber regeneration upon injury. Satellite cell dysfunction has been shown to underlie the …
[HTML][HTML] Negative elongation factor regulates muscle progenitor expansion for efficient myofiber repair and stem cell pool repopulation
Negative elongation factor (NELF) is a critical transcriptional regulator that stabilizes paused
RNA polymerase to permit rapid gene expression changes in response to environmental …
RNA polymerase to permit rapid gene expression changes in response to environmental …
TAK1 modulates satellite stem cell homeostasis and skeletal muscle repair
Satellite cells are resident adult stem cells that are required for regeneration of skeletal
muscle. However, signalling mechanisms that regulate satellite cell function are less …
muscle. However, signalling mechanisms that regulate satellite cell function are less …
MyD88 promotes myoblast fusion in a cell-autonomous manner
Myoblast fusion is an indispensable step for skeletal muscle development, postnatal growth,
and regeneration. Myeloid differentiation primary response gene 88 (MyD88) is an adaptor …
and regeneration. Myeloid differentiation primary response gene 88 (MyD88) is an adaptor …
The IRE1/XBP1 signaling axis promotes skeletal muscle regeneration through a cell non-autonomous mechanism
Skeletal muscle regeneration is regulated by coordinated activation of multiple signaling
pathways. The unfolded protein response (UPR) is a major mechanism that detects and …
pathways. The unfolded protein response (UPR) is a major mechanism that detects and …
Matrix metalloproteinase-9 inhibition improves proliferation and engraftment of myogenic cells in dystrophic muscle of mdx mice
Duchenne muscular dystrophy (DMD) caused by loss of cytoskeletal protein dystrophin is a
devastating disorder of skeletal muscle. Primary deficiency of dystrophin leads to several …
devastating disorder of skeletal muscle. Primary deficiency of dystrophin leads to several …