Oral papilloma lesions may appear as a result of HPV infection, or not, and only special
molecular methods could differentiate them. Low-risk and high-risk HPV types could induce …

The skin is exposed to viral pathogens, but whether they contribute to the oncogenesis of
skin cancers has not been systematically explored. Here we investigated 19 skin tumor …

Three-stranded DNA–RNA structures known as R-loops that form during papillomavirus
transcription can cause transcription–replication conflicts and lead to DNA damage. We …

Countless efforts have been made to eradicate cervical cancer worldwide, including
improving disease screening and human papillomavirus (HPV) vaccination programs …

HPV+ oropharyngeal squamous cell carcinoma (OPC) incidence recently surpassed
cervical cancer and is the most common HPV-related cancer in the developed world. HPV16 …

During the human papillomavirus 16 (HPV16) life cycle, the E2 protein interacts with host
factors to regulate viral transcription, replication, and genome segregation/retention. Our …

The current model of human papillomavirus (HPV) replication is comprised of three modes
of replication. Following infectious delivery, the viral genome is amplified during the …

Human papillomaviruses (HPV) are causative agents in ano-genital and oral cancers;
HPV16 is the most prevalent type detected in human cancers. The HPV16 E6 protein targets …

Beta-human papillomaviruses (HPV) have been implicated in the development of cutaneous
squamous cell cancer (cSCC) in epidermodysplasia verruciformis (EV) patients and organ …

Human papillomavirus (HPV) is a possible carcinogenetic factor in oral squamous cell
carcinoma (OSCC). Previous studies have reported the prevalence of HPV in patients with …