The compound BMAA (β-N-methylamino-L-alanine) has been postulated to play a
significant role in four serious neurological human diseases: Amyotrophic Lateral Sclerosis …

In human neuroblastoma cell cultures, non-human primates and human beings, L-serine is
neuroprotective, acting through a variety of biochemical and molecular mechanisms …

Cyanotoxins are by definition “harmful agents” produced by cyanobacteria. Their toxicity has
been extensively studied and reviewed over the years. Cyanotoxins have been commonly …

Research interest in a non-protein amino acid β-N-methylamino-L-alanine (BMAA) arose
due to the discovery of a connection between exposure to BMAA and the occurrence of …

The neurotoxin β-N-methylamino-l-alanine (BMAA), a non-protein amino acid produced by
terrestrial and aquatic cyanobacteria and by micro-algae, has been suggested to play a role …

Exposure to β-N-methylamino-l-alanine (BMAA) might be linked to the incidence of
amyotrophic lateral sclerosis, Alzheimer's disease and Parkinson's disease. Analytical …

Abstract Anatoxins, saxitoxins, ciguatoxins, beta-N-methylamino-l-alanine and its isomers
are groups of neurotoxins which have a highly specific effect on the nervous system of …

The escalating occurrence of toxic cyanobacterial blooms worldwide is a matter of concern.
Global warming and eutrophication play a major role in the regularity of cyanobacterial …

Cyanobacteria are some of the oldest organisms on earth, and have evolved to produce a
battery of toxic metabolites, including hepatotoxins, dermatoxins, and neurotoxins. In this …

Abstract β-N-methylamino-L-alanine (BMAA) has been shown to accumulate in organisms
by associating with host proteins. It has been proposed that this association is the result of …