Traumatic brain injury leads to cellular damage which in turn results in the rapid release of
damage-associated molecular patterns (DAMPs) that prompt resident cells to release …

Human NK cells can be subdivided into various subsets based on the relative expression of
CD16 and CD56. In particular, CD56 bright CD16−/dim NK cells are the focus of interest …

Sepsis-associated encephalopathy (SAE) is commonly complicated by septic conditions,
and is responsible for increased mortality and poor outcomes in septic patients …

This review recounts the definitions and research evidence supporting the multifaceted roles
of neuroinflammation in the injured brain following trauma. We summarise the literature …

Traumatic brain injury (TBI) is a leading cause of death and disability, and there are currently
no pharmacological treatments known to improve patient outcomes. Unquestionably …

Nosocomial infections are a common occurrence in patients following traumatic brain injury
(TBI) and are associated with an increased risk of mortality, longer length of hospital stay …

Traumatic brain injury (TBI) induces instant activation of innate immunity in brain tissue,
followed by a systematization of the inflammatory response. The subsequent response …

Traumatic brain injury (TBI) and subarachnoid hemorrhage (SAH) are major contributors to
morbidity and mortality. Following the initial insult, patients may deteriorate due to secondary …

BACKGROUND The complex alterations that occur in peripheral immunity after traumatic
brain injury (TBI) have been poorly characterized to date. The purpose of this study was to …

Traumatic brain injury (TBI) is a major cause of mortality and morbidity worldwide. Even
when patients survive the initial insult, there is significant morbidity and mortality secondary …