Diesel exhaust particles (DEP) are known to generate reactive oxygen species in the
respiratory system, triggering cells to activate antioxidant defence mechanisms, such as …

Wildfire smoke induces acute pulmonary distress and is of particular concern to risk groups
such as the sick and elderly. Wood smoke (WS) contains many of the same toxic compounds …

Toxicity of metal nanoparticles (NPs) are closely associated with increasing intracellular
reactive oxygen species (ROS) and the levels of pro-inflammatory mediators. However, NP …

Air pollution has been identified as one of the main environmental risks to health. Since
exercise training seems to act as an anti-inflammatory modulator, our hypothesis is that …

Chronic exposure to ambient levels of air pollution induces respiratory illness exacerbation
by increasing inflammatory responses and apoptotic cells in pulmonary tissues. The …

Diesel exhaust particles (DEPs) from diesel engines produce adverse alterations in cells of
the airways by activating intracellular signaling pathways and apoptotic gene …

We investigate the formation and evolution of silver nanoparticles in the non-irradiated
thermally treated glass system Ag:(37–x) Na 2 O–10CaO–xAl 2 O 3–53SiO 2 mol%, where …

The toxic actions of acute exposition to different diesel exhaust particles (DEPA) fractions on
the mucociliary epithelium are not yet fully understood due to different concentrations of …

Airborne pollutants have detrimental effect on the human body and the environment. Diesel
exhaust particles (DEPs) are known to be major component of particulate matter (PM) and …

Several mechanisms have been suggested to explain the adverse effects of air pollutants on
airway cells. One such explanation is the presence of high concentrations of oxidants and …