Exported proteins of the malaria parasite Plasmodium falciparum interact with proteins of the
erythrocyte membrane and induce substantial changes in the morphology, physiology and …

Plasmodium falciparum, the causative agent of malaria, completely remodels the infected
human erythrocyte to acquire nutrients and to evade the immune system. For this process …

Discovered in 1902 by Georg Maurer as a peculiar dotted staining pattern observable by
light microscopy in the cytoplasm of erythrocytes infected with the human malarial parasite …

Upon invasion of the erythrocyte cell, the malaria parasite remodels its environment; in
particular, it establishes a complex membrane network, which connects the parasitophorous …

The human malarial parasite Plasmodium falciparum exports proteins to destinations within
its host erythrocyte, including cytosol, surface and membranous profiles of parasite origin …

Malaria parasites export proteins beyond their own plasma membrane to locations in the red
blood cells in which they reside. Maurer's clefts are parasite-derived structures within the …

Intracellular parasites from the genus Plasmodium reside and multiply in a variety of cells
during their development. After invasion of human erythrocytes, asexual stages from the …

Presentation of the variant antigen, Plasmodium falciparum erythrocyte membrane protein 1
(EMP1), at knob-like protrusions on the surface of infected red blood cells, underpins the …

Plasmodium falciparum inhabits a niche within the most highly terminally differentiated cell
in the human body–the mature red blood cell. Life inside this normally quiescent cell offers …

Calcium‐dependent protein kinases play a pivotal role in calcium signalling in plants and
some protozoa, including the malaria parasites. They are found in various subcellular …