Alcohol readily crosses the placenta and may disrupt fetal development. Harm from prenatal
alcohol exposure (PAE) is determined by the dose, pattern, timing and duration of exposure …

Drug addiction is a chronic and relapsing brain disorder characterized by compulsive drug
seeking and continued drug use despite adverse consequences. The high prevalence and …

Abstract Purpose of Review There is interest in evaluating the developmental origins of
health and disease (DOHaD) which emphasizes the role of prenatal and early-life …

Prenatal alcohol exposure (PAE) can reprogram the development of cells and tissues,
resulting in a spectrum of physical and neurobehavioral teratology. PAE immediately …

Brain development is a dynamic and lengthy process that includes cell proliferation,
migration, neurogenesis, gliogenesis, synaptogenesis, and pruning. Disruption of any of …

Prenatal alcohol exposure (PAE) impairs recovery from cerebrovascular ischemic stroke in
adult rodents. Since the gut becomes dysbiotic following stroke, we assessed links between …

Fetal alcohol syndrome (FAS) is a developmental and congenital disorder characterized by
neurocognitive impairment, structural defects, and growth restriction due to prenatal alcohol …

Prenatal opioid exposure (POE) is commonly associated with neonatal opioid withdrawal
syndrome (NOWS), which is characterized by a broad variability in symptoms and severity …

Prenatal alcohol exposure (PAE) can have immediate and long-lasting toxic and teratogenic
effects on an individual's development and health. As a toxicant, alcohol can lead to a …

Background Prenatal alcohol exposure (PAE) has been shown to alter fetal blood flow in
utero and is also associated with placental insufficiency and intrauterine growth restriction …