Alzheimer's disease (AD) is a progressive neurodegenerative disease originating partly from
amyloid β protein‐induced synaptic failure. As damaging of noradrenergic neurons in the …

Precise control of norepinephrine (NE) levels and NE-receptor interaction is crucial for
proper function of the brain. Much evidence for this view comes from experimental studies …

Growing evidence suggests that macroautophagy/autophagy-lysosomal pathway deficits
contribute to the accumulation of amyloid-β (Aβ) in Alzheimer disease (AD). Aerobic …

We previously reported that prolonged exposure to an enriched environment (EE) enhances
hippocampal synaptic plasticity, with one of the significant mechanistic pathways being …

Norepinephrine (NE) modulates cognitive function, arousal, attention, and responses to
novelty and stress, and it also regulates neuroinflammation. We previously demonstrated …