AGER1 counteracts cellular oxidant stress induced by AGEs via negative regulation of p66shc-dependent FKHRL1 phosphorylation
W Cai, J He, L Zhu, GE Striker… - Am J Physiol Cell …, 2007 - journals.physiology.org
Advanced glycation end products (AGEs) promote reactive oxygen species (ROS) formation,
and oxidant stress (OS) in diabetes and aging-related diseases. AGE-induced OS is …
and oxidant stress (OS) in diabetes and aging-related diseases. AGE-induced OS is …
Advanced glycation end product (AGE) receptor 1 suppresses cell oxidant stress and activation signaling via EGF receptor
W Cai, JC He, L Zhu, C Lu… - Proceedings of the …, 2006 - National Acad Sciences
Advanced glycation end product receptors (AGERs) play distinct functional roles in both the
toxicity and disposal of advanced glycation end products (AGEs), substances that are linked …
toxicity and disposal of advanced glycation end products (AGEs), substances that are linked …
[引用][C] AGER1 overexpression protects against acute kidney injury by suppressing endoplasmic reticulum stress, a new mechanism of renal AGE-toxicity
R Zhang, M Torreggiani, W Cai… - …, 2008 - AMER DIABETES ASSOC 1701 N …
Early Growth Response 1 Contributes to Renal IR Injury by Inducing Proximal Tubular Cell Apoptosis
K Jeong, J Je, T Dusabimana, H Kim… - International Journal of …, 2023 - mdpi.com
Renal ischemia–reperfusion (IR) causes acute kidney injury due to oxidative stress, tubular
inflammation, and apoptosis. Early growth response 1 (Egr-1) is a transcription factor …
inflammation, and apoptosis. Early growth response 1 (Egr-1) is a transcription factor …
Advanced glycation end product receptor-1 transgenic mice are resistant to inflammation, oxidative stress, and post-injury intimal hyperplasia
M Torreggiani, H Liu, J Wu, F Zheng, W Cai… - The American journal of …, 2009 - Elsevier
The high levels of oxidative stress (OS) and inflammation associated with cardiovascular
disease are linked to pro-oxidants such as advanced glycation end products (AGEs). AGEs …
disease are linked to pro-oxidants such as advanced glycation end products (AGEs). AGEs …
AGER1 regulates endothelial cell NADPH oxidase-dependent oxidant stress via PKC-δ: implications for vascular disease
W Cai, M Torreggiani, L Zhu, X Chen… - … of Physiology-Cell …, 2010 - journals.physiology.org
Advanced glycated end-product receptor 1 (AGER1) protects against vascular disease
promoted by oxidants, such as advanced glycated end products (AGEs), via inhibition of …
promoted by oxidants, such as advanced glycated end products (AGEs), via inhibition of …
AGE-receptor-1 counteracts cellular oxidant stress induced by AGEs via negative regulation of p66shc-dependent FKHRL1 phosphorylation
W Cai, JC He, L Zhu, X Chen… - American Journal of …, 2008 - journals.physiology.org
Advanced glycation end products (AGEs) promote reactive oxygen species (ROS) formation
and oxidant stress (OS) in diabetes and aging-related diseases. AGE-induced OS is …
and oxidant stress (OS) in diabetes and aging-related diseases. AGE-induced OS is …
Receptor for AGEs (RAGE) blockade may exert its renoprotective effects in patients with diabetic nephropathy via induction of the angiotensin II type 2 (AT2) receptor
KC Sourris, AL Morley, A Koitka, P Samuel… - Diabetologia, 2010 - Springer
Aims/hypothesis The receptor for AGEs (RAGE) contributes to the development and
progression of diabetic nephropathy. In this study, we examined whether the protective …
progression of diabetic nephropathy. In this study, we examined whether the protective …
Erianin protects against high glucose-induced oxidative injury in renal tubular epithelial cells.
CMF Chen MeiFen, LSS Liou ShorongShii… - 2019 - cabidigitallibrary.org
Erianin is the major bibenzyl compound found in Dendrobium chrysotoxum Lindl. The
current study was designed to investigate the protective effects of erianin on high glucose …
current study was designed to investigate the protective effects of erianin on high glucose …
Involvement of endoplasmic reticulum stress, autophagy and apoptosis in advanced glycation end products-induced glomerular mesangial cell injury
CK Chiang, CC Wang, TF Lu, KH Huang, ML Sheu… - Scientific Reports, 2016 - nature.com
Advanced glycation end-products (AGEs)-induced mesangial cell death is one of major
causes of glomerulus dysfunction in diabetic nephropathy. Both endoplasmic reticulum (ER) …
causes of glomerulus dysfunction in diabetic nephropathy. Both endoplasmic reticulum (ER) …